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The result of a genetic test does not indicate the current health status. Detecting a genetic mutation is not equivalent with neoplasm development at a given moment. It proves increased predisposition, a predisposition for developing neoplasm at a much younger age than the population average, and therefore, it indicates that there is a need to introduce appropriate prophylaxis. 

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When it comes to cancer diagnosis, speed and accuracy make all the difference. Thanks to our cancer panels, the diagnoses may be faster and more effective than it was ever before.

The DNA Research Center is a world-class research facility that will revolutionize the way we diagnose and treat cancer. The Centre as the first one in Poland offers three cancer panels targeting genes previously linked to a genetic predisposition towards cancer.

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902 Breast cancer and ovary cancer – analysis of 5 mutations of BRCA1 gene

Gene BRCA1 is responsible in a cell for composition of protein, which repairs damages and helps retaining genetic information in the unchanged form in a cell. The test covers the analysis of 5 most frequent mutations of gene BRCA1 responsible for development of genetically conditioned breast and ovary cancers: C61G, 4153delA, 5382insC, 3819del5, C64R, which account for ca. 90% of mutations identified in that gene.

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916 Genetic predisposition for breast cancer and prostate cancer – analysis of mutations of gene NBS1 (NBN)

Protein NBN, just like BRCA1, helps to repair damaged genetic information in cells. The test detects carrier state of mutation 657del5 in gene NBS1. Homozygous carriage (all copies of a gene with mutation) 657del5 is responsible for a genetic disease, the so called Nijmegen breakage syndrome, NBS, which is characterized, among others, by increased risk of developing neoplasm. While the fact of carrying mutation 657del5 increases by ca. 2 times the risk of developing breast cancer and by ca. 4 times prostate cancer, as compared to people without the mutation.

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